Volume 128, Issue 10 pp. 3499-3502
Zuschrift

Drug Repurposing Identifies Inhibitors of Oseltamivir-Resistant Influenza Viruses

Dr. Ju Bao

Dr. Ju Bao

Department of Structural Biology, St. Jude Children's Research Hospital, Memphis, TN, 38105 USA

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Dr. Bindumadhav Marathe

Dr. Bindumadhav Marathe

Department of Infectious Diseases, St. Jude Children's Research Hospital, Memphis, TN, 38105 USA

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Dr. Elena A. Govorkova

Dr. Elena A. Govorkova

Department of Infectious Diseases, St. Jude Children's Research Hospital, Memphis, TN, 38105 USA

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Prof. Jie J. Zheng

Corresponding Author

Prof. Jie J. Zheng

Department of Structural Biology, St. Jude Children's Research Hospital, Memphis, TN, 38105 USA

Stein Eye Institute, Department of Ophthalmology, David Geffen School of Medicine at UCLA, Los Angeles, CA, 90095 USA

Department of Infectious Diseases, St. Jude Children's Research Hospital, Memphis, TN, 38105 USA

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First published: 02 February 2016
Citations: 1

Abstract

The neuraminidase (NA) inhibitor, oseltamivir, is a widely used anti-influenza drug. However, oseltamivir-resistant H1N1 influenza viruses carrying the H275Y NA mutation spontaneously emerged as a result of natural genetic drift and drug treatment. Because H275Y and other potential mutations may generate a future pandemic influenza strain that is oseltamivir-resistant, alternative therapy options are needed. Herein, we show that a structure-based computational method can be used to identify existing drugs that inhibit resistant viruses, thereby providing a first line of pharmaceutical defense against this possible scenario. We identified two drugs, nalidixic acid and dorzolamide, that potently inhibit the NA activity of oseltamivir-resistant H1N1 viruses with the H275Y NA mutation at very low concentrations, but have no effect on wild-type H1N1 NA even at a much higher concentration, suggesting that the oseltamivir-resistance mutation itself caused susceptibility to these drugs.

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