Genes, Chromosomes and Cancer: Vol 51, No 5

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Genes, Chromosomes and Cancer

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Genes, Chromosomes and Cancer: Volume 51, Issue 5

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From cryptic chromosomal lesions to pathologically relevant genes: Integration of SNP-array with gene expression profiling in myelodysplastic syndrome with normal karyotype

Mitotic recombination of chromosome arm 17q as a cause of loss of heterozygosity of NF1 in neurofibromatosis type 1-associated glomus tumors ^†

The impact of the genetic background on the genome make-up of tumor cells

Internal tumor burden in neurofibromatosis Type I patients with large NF1 deletions

Identification of TFG (TRK-fused gene) as a putative metastatic melanoma tumor suppressor gene

Contribution of germline mutations to PARK2 gene inactivation in lung adenocarcinoma ^†

The microRNAs, MiR-31 and MiR-375, as candidate markers in Barrett's esophageal carcinogenesis

Somatic mutations in the notch, NF-KB, PIK3CA, and hedgehog pathways in human breast cancers

A new whole genome amplification method for studying clonal evolution patterns in malignant colorectal polyps ^†^‡

Localized DNA cleavage secondary to genotoxic exposure adjacent to an Alu inverted repeat

Fusion of the AHRR and NCOA2 genes through a recurrent translocation t(5;8)(p15;q13) in soft tissue angiofibroma results in upregulation of aryl hydrocarbon receptor target genes

Letters to the Editors

Allelic drop-out, allele imbalance, or loss of heterozygosity?

Letters to the Editor

Reply to Kluwe “Allelic Drop-Out, Allele Imbalance, or Loss of Heterozygosity?”

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Genes, Chromosomes and Cancer

Journal list menu

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Genes, Chromosomes and Cancer: Volume 51, Issue 5

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Research Articles

From cryptic chromosomal lesions to pathologically relevant genes: Integration of SNP-array with gene expression profiling in myelodysplastic syndrome with normal karyotype

Mitotic recombination of chromosome arm 17q as a cause of loss of heterozygosity of NF1 in neurofibromatosis type 1-associated glomus tumors†

The impact of the genetic background on the genome make-up of tumor cells

Internal tumor burden in neurofibromatosis Type I patients with large NF1 deletions

Identification of TFG (TRK-fused gene) as a putative metastatic melanoma tumor suppressor gene

Contribution of germline mutations to PARK2 gene inactivation in lung adenocarcinoma†

The microRNAs, MiR-31 and MiR-375, as candidate markers in Barrett's esophageal carcinogenesis

Somatic mutations in the notch, NF-KB, PIK3CA, and hedgehog pathways in human breast cancers

A new whole genome amplification method for studying clonal evolution patterns in malignant colorectal polyps†‡

Localized DNA cleavage secondary to genotoxic exposure adjacent to an Alu inverted repeat

Fusion of the AHRR and NCOA2 genes through a recurrent translocation t(5;8)(p15;q13) in soft tissue angiofibroma results in upregulation of aryl hydrocarbon receptor target genes

Letters to the Editors

Allelic drop-out, allele imbalance, or loss of heterozygosity?

Letters to the Editor

Reply to Kluwe “Allelic Drop-Out, Allele Imbalance, or Loss of Heterozygosity?”

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More from this journal

Mitotic recombination of chromosome arm 17q as a cause of loss of heterozygosity of NF1 in neurofibromatosis type 1-associated glomus tumors ^†

Contribution of germline mutations to PARK2 gene inactivation in lung adenocarcinoma ^†

A new whole genome amplification method for studying clonal evolution patterns in malignant colorectal polyps ^†^‡