Reasons to Publish in Traffic

  • New Editorial Team in 2025
  • Run by active scientists in the cell biology community.
  • Constructive and transparent peer review: editorial correspondence published with accepted articles through Publons (but referees remain anonymous).
  • Fast editorial process, an average of 23 days from submission to first decision.
  • All contents available free online after 6 months
  • Authors can submit their work at initial submission in the format of their choice

Overview

Traffic covers the cell biology and biochemistry of intracellular transport in health and disease and aims to publish manuscripts at the forefront of this field. The aim of Traffic is to publish papers that provide insight into the mechanisms that underlie subcellular trafficking, membrane dynamics, and organelle function and how these subcellular processes influence cellular physiology. Specific topics are outlined in the Aims & Scope below.

The Editors of Traffic have signed up to The San Francisco Declaration on Research Assessment (DORA). For more information please visit the DORA website.

Traffic publishes the editorial correspondence related to the review of accepted papers as supplemental material online. This includes the decision letter(s), referee comments and author rebuttals. Referee identities and confidential comments to the editor will remain confidential.

Starting in March 2019, all review documents will be hosted on Publons and linked to from the published article. 

Aims and Scope

Traffic encourages and facilitates the publication of papers in any field relating to intracellular transport in health and disease. Traffic papers span disciplines such as developmental biology, neuroscience, innate and adaptive immunity, epithelial cell biology, intracellular pathogens and host-pathogen interactions, among others using any eukaryotic model system. Areas of particular interest include protein, nucleic acid and lipid traffic, molecular motors, intracellular pathogens, intracellular proteolysis, nuclear import and export, cytokinesis and the cell cycle, the interface between signaling and trafficking or localization, protein translocation, the cell biology of adaptive an innate immunity, organelle biogenesis, metabolism, cell polarity and organization, and organelle movement.

All aspects of the structural, molecular biology, biochemistry, genetics, morphology, intracellular signaling and relationship to hereditary or infectious diseases will be covered. Manuscripts must provide a clear conceptual or mechanistic advance. The editors will reject papers that require major changes, including addition of significant experimental data or other significant revision.

Traffic will consider manuscripts of any length, but encourages authors to limit their papers to 16 typeset pages or less.

 

Traffic发表探讨细胞内运输在健康和疾病中作用的文章。涉及的学科领域包括发育生物学，神经科学，先天和适应性免疫，上皮细胞生物学，细胞内病原体和宿主-病原体相互作用，对任何真核模型系统的使用。特别感兴趣的领域包括蛋白质，核酸和脂类运输，分子马达，细胞内病原体，细胞内蛋白质降解，细胞核物质的输入和输出，胞质分裂和细胞周期，细胞信号和细胞运输的交叉领域，蛋白质转运，适应性先天免疫细胞生物学，细胞器生源论，细胞极性和组织，细胞器的运动。

细胞结构、分子生物学、生物化学、遗传学、形态学、细胞内信号与遗传或感染性疾病的关系的所有方面都将涉及。稿件必须提供一个清晰的概念或机制的进展。编辑看到需要大修的论文会直接拒稿，包括需要增加重要的实验数据或其他重大修改。

Traffic对稿件的长度没有特定要求，但文章最好不要多于16个版面。

 

Keywords

actin, adipocyte cell biology, antigen processing and presentation, autophagy, biosynthetic pathway, cell adhesion, cell modeling, cell morphogenesis, chloroplast assembly, chloroplast protein export, chloroplast protein import, chloroplasts, cilia, cilia assembly, ciliary function, cytokinesis, cytoskeletal function, development, endocytosis, endoplasmic reticulum, endosomal recycling, endosomes, ERGIC, G-protein coupled receptors, glycolipid assembly and trafficking, glycoprotein assembly and trafficking, Golgi complex, hereditary diseases of membrane trafficking, immune cell biology, immunity, inflammation, intracellular motility, intracellular pathogens, intracellular proteolysis, lipid bodies, lipid droplets, lipid microdomains, lipid trafficking, lysosomal storage diseases, lysosome-related organelles, lysosomes, membrane dynamics, membrane fission, membrane fusion, membrane trafficking, microfilaments, microtubule motors, microtubules, mitochondria, mitochondrial fission and fusion, mitochondrial protein import, model organisms, model systems, muscle cell biology, myosins, neurons, nuclear export, nuclear import, nuclear lamina, nuclear structure, nuclear transport, nucleus, organelle biogenesis, organelle motility, parasite cell biology, peroxisome assembly, peroxisome protein import, phagocytosis, phagosome maturation, plastids, protein complex assembly, protein folding, protein structure, protein trafficking, protein translocation, quality control, rab proteins, receptor down-regulation, regulated secretion, secretion, secretory granules, secretory packages, signaling, small G-proteins, subcellular organelles, synaptic vesicles, vacuole, vesicle, vesicular transport carriers, viral trafficking, virus assembly, virus cell biology, virus entry

 

 

Abstracting and Indexing Information

 

  • Academic Search (EBSCO Publishing)
  • Academic Search Alumni Edition (EBSCO Publishing)
  • Academic Search Premier (EBSCO Publishing)
  • AGRICOLA Database (National Agricultural Library)
  • Biological Science Database (ProQuest)
  • Current Contents: Life Sciences (Clarivate Analytics)
  • Embase (Elsevier)
  • Journal Citation Reports/Science Edition (Clarivate Analytics)
  • MEDLINE/PubMed (NLM)
  • Natural Science Collection (ProQuest)
  • PubMed Dietary Supplement Subset (NLM)
  • Research Alert (Clarivate Analytics)
  • Science Citation Index Expanded (Clarivate Analytics)
  • SciTech Premium Collection (ProQuest)
  • SCOPUS (Elsevier)