Volume 59, Issue 4 pp. 455-457
Original Articles: Hepatology and Nutrition

Vitamin and Zinc Status Pretreatment and Posttreatment in Patients With Inflammatory Bowel Disease

Neha R. Santucci

Corresponding Author

Neha R. Santucci

Department of Pediatrics, SUNY at Buffalo, Digestive Diseases and Nutrition Center, Children's Hospital of Buffalo, New York

Address correspondence and reprint requests to Razan H. Alkhouri, MD, and Neha R. Santucci, Department of Pediatrics, SUNY at Buffalo, Digestive Diseases and Nutrition Center, 219 Bryant Street, Buffalo, NY 14222 (e-mail: [email protected] and [email protected]).Search for more papers by this author
Razan H. Alkhouri

Corresponding Author

Razan H. Alkhouri

Department of Pediatrics, SUNY at Buffalo, Digestive Diseases and Nutrition Center, Children's Hospital of Buffalo, New York

Address correspondence and reprint requests to Razan H. Alkhouri, MD, and Neha R. Santucci, Department of Pediatrics, SUNY at Buffalo, Digestive Diseases and Nutrition Center, 219 Bryant Street, Buffalo, NY 14222 (e-mail: [email protected] and [email protected]).Search for more papers by this author
Robert D. Baker

Robert D. Baker

Department of Pediatrics, SUNY at Buffalo, Digestive Diseases and Nutrition Center, Children's Hospital of Buffalo, New York

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Susan S. Baker

Susan S. Baker

Department of Pediatrics, SUNY at Buffalo, Digestive Diseases and Nutrition Center, Children's Hospital of Buffalo, New York

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First published: 01 October 2014
Citations: 34

The authors report no conflicts of interest.

ABSTRACT

Vitamin deficiencies are common in inflammatory bowel disease. Here we present 5-year follow-up data of 61 patients. No folate or vitamin B12 deficiency was identified throughout the study. A daily multivitamin supplement was sufficient to replete 100% of vitamin A–deficient and vitamin E–deficient patients. A total of 52% of vitamin D–deficient patients corrected, but 15% who had normal vitamin D levels at diagnosis developed deficiency. A total of 63% of zinc-deficient patients normalized their zinc status, but 15% developed zinc deficiency at follow-up despite supplementation.

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