Centre for Cell Biology and Cutaneous Research, Blizard Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, London, E1 2AT UK
Centre for Cell Biology and Cutaneous Research, Blizard Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, London, E1 2AT UK
1Hara M, Verkman AS. Glycerol replacement corrects defective skin hydration, elasticity, and barrier function in aquaporin-3-deficient mice. Proc Natl Acad Sci U S A2003; 100: 7360–5.
3Luo J, Liu X, Liu Jet al. Activation of TGF-β1 by AQP3-mediated H2O2 transport into fibroblasts of a bleomycin-induced mouse model of scleroderma. J Invest Dermatol2016; 136: 2372–9.
4Farhadi E, Mahmoudi M, Rahmani Fet al. Attenuation of aquaporin-3 and epidermal growth factor receptor expression and activation in systemic sclerosis dermal fibroblasts. J Cell Physiol2019; 234: 12876–83.
6Ding S, Chen J, Zeng Qet al. Chronic sun exposure is associated with distinct histone acetylation changes in human skin. Br J Dermatol2018; 179: 110–17.
7Choudhary V, Olala LO, Kagha Ket al. Regulation of the glycerol transporter, aquaporin-3, by histone deacetylase-3 and p53 in keratinocytes. J Invest Dermatol2017; 137: 1935–44.
9Majora M, Sondenheimer K, Knechten Met al. HDAC inhibition improves autophagic and lysosomal function to prevent loss of subcutaneous fat in a mouse model of Cockayne syndrome. Sci Transl Med2018; 10: eaam7510.
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