Volume 37, Issue 4 1 pp. 911-914
Article

Observation of Vocal Fold and Pharyngeal Paralysis After Carotid Endarterectomy Using a Magnifying Laryngoscope

Tomonori Tamaki

Corresponding Author

Tomonori Tamaki

Department of Neurosurgery, Nippon Medical School, Tamanagayama Hospital, 1-7-1 Nagayama, Tamashi, 206-8512 Tokyo, Japan

[email protected]Search for more papers by this author
Yoji Node

Yoji Node

Department of Neurosurgery, Nippon Medical School, Tamanagayama Hospital, 1-7-1 Nagayama, Tamashi, 206-8512 Tokyo, Japan

Search for more papers by this author
Norihiro Saitou

Norihiro Saitou

Department of Neurosurgery, Asahikawa Red Cross Hospital, 1-1-1 Akebonoichijyo, 070-8530 Asahikawashi, Hokkaido, Japan

Search for more papers by this author
Hideto Saigusa

Hideto Saigusa

Department of Otolaryngology, Nippon Medical School, 1-1-5 Sendagi, Bunkyo-ku, 113-8603 Tokyo, Japan

Search for more papers by this author
First published: 29 January 2013
Citations: 6

Abstract

Background

Injury to the vagus nerve or one of its branches during carotid endarterectomy (CEA) can result in vocal fold paralysis (VFP), but the exact mechanisms and site of injury responsible for VFP after CEA are unclear. The aim of this study was to identify the site of nerve injury in patients with VFP after CEA using magnifying laryngoscopy.

Methods

We performed 96 consecutive CEA procedures in 87 patients over 5 years. After 56 CEA procedures, we examined vocal fold movements with a flexible nasolaryngoscope and detected VFP in 5 of 40 cases (9 %). At 6–8 weeks after CEA, these five patients also underwent magnifying laryngoscopy at another institution by a specialist in vocalization.

Results

We confirmed ipsilateral VFP and pharyngeal paralysis in three patients. The other two patients recovered from their nerve injuries spontaneously before the magnifying examination.

Conclusions

VFP and pharyngeal paralysis were caused by damage to the recurrent laryngeal and pharyngeal nerves. Therefore, the probable site of nerve injury during CEA was near the inferior vagal ganglion of the vagus nerve trunk in our three patients.

The full text of this article hosted at iucr.org is unavailable due to technical difficulties.